Blood gases & acid-base

ABG interpreter

Enter three numbers off the gas and walk the classic stepwise method — disorder, compensation, gap, delta — with the mixed pictures unmasked.

FreeNo sign-upWorks offlineNo patient data stored
pH
pCO₂
kPa
HCO₃
mmol/L
Sodium (optional)
mmol/L
Chloride (optional)
mmol/L
Albumin (optional)
g/L
PaO₂ (optional)
kPa
FiO₂ (optional)
%
Compensation: Winter's (metabolic acidosis), 0.7×ΔHCO₃ (metabolic alkalosis), 1/4–5 per 10 mmHg pCO₂ (respiratory). References: Winter 1967; Berend et al., NEJM 2014.Anion gap corrected +0.25 per g/L albumin below 40. Computed outputs report mmHg by convention (÷7.5 for kPa).A–a gradient assumes sea level (760 mmHg) — on the Highveld (~1600 m) it reads roughly 20–25 mmHg high; interpret against local norms.Interpret alongside the clinical picture — no algorithm replaces it.
Worked example

A patient in DKA: pH 7.25, pCO₂ 3.3 kPa (24.8 mmHg), HCO₃ 10 mmol/L, sodium 140 and chloride 100 mmol/L, PaO₂ 8.0 kPa (60 mmHg) on room air.

Anion gap 30 mmol/L, expected pCO₂ 23 mmHg, delta ratio 1.29 — high-anion-gap metabolic acidosis, compensation appropriate

A pure high-anion-gap acidosis: the measured pCO₂ sits where Winter's formula predicts, so there is no hidden respiratory disorder — a pCO₂ drifting above the expected value would flag a tiring patient about to decompensate.

Who this is for

For clinicians interpreting arterial blood gases in the emergency centre, ICU, or ward — from DKA and sepsis to chronic CO₂ retention. Enter pH, pCO₂, and HCO₃ (kPa first, as South African analysers report) and the stepwise method runs: primary disorder and compensation checking. Add sodium, chloride, and albumin for the corrected anion gap and delta ratio, and oxygen values for the A–a gradient and P/F ratio.

How it works

What it does

A stepwise arterial blood gas interpretation: identifies the primary acid-base disorder, checks whether compensation is appropriate (Winter's formula and its counterparts), computes the anion gap with albumin correction, the delta ratio, and — when oxygen values are supplied — the A–a gradient and P/F ratio.

Why compensation checking matters

The compensation rules are what unmask mixed disorders: a DKA patient whose pCO₂ is higher than Winter's formula predicts has a concurrent respiratory acidosis, often a tiring patient about to decompensate.

Units

Defaults to kPa as South African blood gas analysers report; toggle to mmHg. The compensation formulas run in mmHg internally.

Built on published evidence

  • Compensation: Winter's (metabolic acidosis), 0.7×ΔHCO₃ (metabolic alkalosis), 1/4–5 per 10 mmHg pCO₂ (respiratory). References: Winter 1967; Berend et al., NEJM 2014

Codification last reviewed 2026-08-28, and checked against published reference values in automated tests on every release — see the full evidence ledger.

Questions clinicians ask

Does it take kPa or mmHg?

Both — kPa is the default, matching South African blood gas analysers, with mmHg one tap away. Computed outputs such as the expected pCO₂ and A–a gradient report in mmHg by convention (divide by 7.5 for kPa); the compensation formulas run in mmHg internally.

What does the expected pCO₂ actually tell me?

Whether compensation is appropriate — Winter's formula in metabolic acidosis, with counterparts for the other disorders (Berend, NEJM 2014). A measured pCO₂ off the expected value unmasks a second, concurrent disorder: in DKA, a pCO₂ above Winter's prediction means a respiratory acidosis is hiding in the picture.

Why correct the anion gap for albumin?

Hypoalbuminaemia lowers the apparent gap — the correction adds 0.25 mmol/L per g/L of albumin below 40, so a significant acidosis is not concealed behind an 'unremarkable' gap in an unwell, low-albumin patient.

Is the A–a gradient valid at altitude?

The calculation assumes sea level (760 mmHg), so on the Highveld at around 1600 m it reads roughly 20–25 mmHg high — interpret it against local norms rather than sea-level cut-offs.

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